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For research purposes onlyThese compounds aren't FDA approved. All data presented is from clinical trials for educational reference.

Tesamorelin

4.9 (157)

Growth Hormone Releasing Peptide

Dosage

Quantity

1

Price

$62.99

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Made in the USA

Certificate of Analysis

Batch verified lab data

Latest

99.93%

Purity

Variant
Tesamorelin 10mg
Lot #
A0112
Labeled
20mg
Actual
22.56mg
Tested
Feb 4, 2026

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Research Purposes Snapshot

Summarizes published clinical and metabolic data from the tesamorelin development program (EGRIFTA) in HIV-associated lipodystrophy and related analyses. Educational only—does not replace FDA labeling or clinician judgment.

p = 0.005

Executive function

Significant cognitive score improvement reported in a subset analysis (trial-dependent endpoints).

18.2%

Visceral fat

Mean VAT reduction vs baseline at ~26 weeks in pooled Phase 3 HIV lipodystrophy trials.

117%

IGF-1 rise

Marked IGF-1 elevation versus baseline in monitored cohorts (lab-dependent).

37%

Hepatic fat

Liver fat fraction improvements at ~12 months in NAFLD substudies.

35%

NAFLD resolution

Complete fatty-liver resolution endpoint in a subset of responders.

Mechanism of action (review context)

GHRH receptor engagement

Synthetic GHRH analog binds pituitary GHRH receptors to restore pulsatile GH output with intact feedback loops.

IGF-1–linked lipolysis

Hepatic IGF-1 generation supports preferential visceral adipose loss while lean mass is largely preserved in trial narratives.

Metabolic remodeling

Ectopic fat reduction in liver and muscle correlates with improved insulin sensitivity markers without destabilizing mean glucose in many reports.

Differentiator vs exogenous GH: preserves endogenous pulsatile GH secretion rather than continuous pharmacologic GH exposure.

LIPO-010 & CTR-1011

18.2%

Average visceral adipose reduction at 26 weeks

Tesamorelin — week 26 VAT change−15.4%
Tesamorelin — week 52 VAT change−17.5%
Placebo−5%

816 HIV-positive adults with lipodystrophy; primary endpoint = percent trunk fat (VAT) change by CT at 26 weeks vs placebo.

Consult FDA labels and primary trial manuscripts for full inclusion criteria.

Treatment outcomes

Responder and tolerability signals

Subjects with >8% VAT reduction (primary responders)73%
Completed planned therapy (tolerability narrative)91.2%
Durability: benefit narratives extend through 52 weeks with continued dosing in extension analyses.

VAT trajectory

Active drug vs placebo (illustrative)

Week 26 (−15.4%)
Week 52 (−17.5%)
Placebo (−5%)

IGF-1 axis

108–117%

Mean vs peak elevations vary by cohort and assay timing.

Triglycerides

−20

mg/dL-type reductions in VAT responders (range varies).

Lean mass

Preserved

No meaningful lean-tissue loss in pivotal narratives.

Body image

Improved

Patient-reported outcome shifts in QoL instruments.

Metabolic markers

Lipids & glucose

  • Triglycerides↓ 20–50 mg/dL
  • Total cholesterol↓ ~9 mg/dL
  • Adiponectin↑ ~12%
  • Fasting glucoseNo major mean shift

Muscle imaging

Density & cross-sectional area

  • Trunk muscle density+4.86 HU
  • Psoas cross-sectional area+0.46 cm²
  • Rectus cross-sectional area+0.44 cm²

Absolute imaging units (not percent bars)—see trial imaging supplements.

Hepatic readouts

NAFLD / NASH signals

  • Hepatic fat ↓ ~37% in substudy cohorts
  • Fibrosis scores trend favorable in exploratory analyses
  • Transcriptomic shifts toward reduced steatohepatitis risk

EGRIFTA remains the only FDA-approved therapy specifically for HIV-associated excess visceral adiposity with linked hepatic benefit discussions—verify current label indications.

Safety profile (Phase 3, n≈816)

GH-axis–related effects are expected and usually mild–moderate.

25%

Injection site reactions

Mild
13.3%

Arthralgia

Mild
7.5%

Peripheral edema

Mild
5.7%

Myalgia

Mild
5.1%

Paresthesia

Mild
4%

Hypersensitivity

Moderate

Anti-drug antibodies

Binding antibodies were detected in a subset yet did not blunt VAT loss or IGF-1 increments in aggregate analyses—clinical relevance judged low.

Discontinuation drivers

  • Any AE-related stop8.8%
  • GH-related events4.2%
  • Injection reactions4.6%
  • Serious AEs<4%

Most discontinuations clustered in the first 26 weeks.

Labeled warnings context

  • Glucose intolerance / diabetes vigilance (~5% HbA1c ≥6.5% signal in summaries)
  • Theoretical malignancy vigilance with sustained IGF-1 elevation
  • Diabetic retinopathy progression risk in susceptible patients
  • Fluid retention and edema in predisposed cohorts

Storage handling reference

Peptide handling

Cold chain

Follow manufacturer stability data.

Reconstitution

Sterile technique only.

Light

Protect from photodegradation.

Documentation

Log batch, date, and temperature.

Researcher notes

  • Contraindicated with active malignancy per prescribing information—mirror that caution in any research plan.
  • Use extra glycemic surveillance in patients with diabetes or prediabetes phenotypes.
  • Monitor IGF-1 on a schedule aligned with investigator protocols and local regulations.

Important Research Notice

Not for human consumption. This product and all products are sold exclusively for research and educational purposes. It is not intended to diagnose, treat, cure, or prevent any disease.

All clinical trial data and research findings presented on this page are sourced from journals and official publications but should be fact checked. They are provided for educational reference only and should not be interpreted as medical advice or product claims.

By purchasing this product, you confirm that you are a qualified researcher and will use it in accordance with all applicable laws and regulations and you do not intend to use it for human consumption.